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#181
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Re: 27,2 kg thủy ngân phát tán ra môi trường, kiến nghị tẩy độc khu vực Nhà máy Rạng Đông
Khi nào có người nằm giãy ra chết thì sai đến đâu chúng tôi xử đến đó nhé
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| cutilua3003 |
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#182
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Re: Page 19 - 27,2 kg thủy ngân phát tán ra môi trường, kiến nghị tẩy độc khu vực Nhà máy Rạng Đông
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| Hanoikute5 |
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#183
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Re: 27,2 kg thủy ngân phát tán ra môi trường, kiến nghị tẩy độc khu vực Nhà máy Rạng Đông
__________________
\m/ the show must go on \m/ |
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#184
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Re: 27,2 kg thủy ngân phát tán ra môi trường, kiến nghị tẩy độc khu vực Nhà máy Rạng Đông
Nguy hiểm quá. Thêm vài thông tin để ai có bị thì đọc biết. Tóm tắt là bị một loạt vấn đề liên quan đến cấp tính và mãn tính: Phổi, tổn thương não, hư thận,...
![]() https://rais.ornl.gov/tox/profiles/mercury_f_V1.html 3.2. INHALATION EXPOSURES 3.2.1. Acute Toxicity 3.2.1.1. Human Inhalation of mercury vapor may result in corrosive bronchitis, interstitial pneumonitis, and death (Goyer 1991). Systemic effects following inhalation exposure may include shock, renal disorders, and central nervous system effects characterized by lethargy and neurobehavioral effects (insomnia, loss of memory, excitability, etc.). Occupational exposure to metallic mercury vapor at concentrations of 1.1 to 44 mg/m3 for 4 to 8 hours produced chest pains, dyspnea, cough, hemoptysis, impairment of pulmonary function, and interstitial pneumonitis (ATSDR 1989). Acute effects of inorganic mercury poisoning may be accompanied by a metallic taste, sore gums, and excessive salivation. A case report cited an incident wherein four adults were acutely exposed to mercury vapor resulting from the smelting of dental amalgams (Taueg et al. 1991). Initial signs of toxicity included nausea, diarrhea, dyspnea, and chest pains. Despite chelation therapy, all four patients died 11 to 24 days after initial exposure. Mercury concentrations in the house were as high as 912 µg/m3 at or within 11 to 188 days after the exposure, and postmortem blood mercury levels ranged from 58 to 369 µg/L. Historically, the triad of increased excitability, tremors, and gingivitis has been recognized as characteristic for mercury poisoning (Goyer 1991). 3.2.1.2. Animal Death resulting from severe pulmonary edema has been reported for mice, guinea pigs, and rats following inhalation exposure to mercury vapor (Christensen et al. 1937). Similarly, inhalation exposure of rabbits to mercury vapor at a concentration of 1 to 1.1 mg/m3 for 1 to 30 hours resulted in death (Ashe et al. 1953). This same study also showed that 30-hour exposure of rabbits to mercury vapor at a concentration of 28.8 mg/m3 caused extensive necrosis of the lungs. Data are lacking regarding the effects of inhalation exposure of animals to organic mercury compounds (ATSDR 1989). 3.2.2. Subchronic Toxicity 3.2.2.1. Human Subchronic inhalation exposure to mercury vapor will result in effects similar to those for acute exposure and will vary depending on exposure severity and duration. Sax and Lewis (1989) reported a lowest toxic exposure level of 0.15 mg/m3 for human females exposed to mercury vapor for 46 days. Sexton et al. (1976) reported tremors (especially in activities requiring fine control), insomnia, and nervousness resulting from 7 to 25 weeks of exposure to mercury vapor. Langolf et al. (1978) noted that short-term exposure to high levels of mercury appears to induce greater neurological effects than does long-term exposure to lower mercury levels. Exposure of humans to diethylmercury at vapor concentrations of 1 to 1.1 mg/m3 for 4 to 5 months resulted in death, the cause of which was not determined (Hill 1943). Data are lacking regarding inhalation exposure to methyl mercury. Exposure of female workers to mercury vapor (<0.02 mg Hg/m3, 8 hrs/day, 44 hrs/week) for 23 months did not produce any signs or symptoms of toxicity (Ishihara and Urushiyama 1994). 3.2.2.2. Animal The effects of subchronic inhalation exposure to mercury or mercury compounds is dependent on the exposure concentration and the specific form of mercury. Low levels of exposure will generally affect the kidney and central nervous system while high-level exposure will target the respiratory, cardiovascular, and gastrointestinal systems as described in Sect. 3.1. Exposure of rabbits to mercury vapor (0.86 to 6.0 mg/m3) for 2 to 12 weeks resulted in marked degeneration and necrosis of the heart (Ashe et al. 1953). Subchronic inhalation exposure of rats and rabbits to mercury has also produced neurobehavioral changes (ATSDR 1989). Evidence for a systemic autoimmune response was reported by Bernaudin et al. (1981) for rats inhaling vapors of mercuric chloride or methyl mercuric chloride 4 hours/day for 60 days. The kidney, lungs, and spleen were identified as target organs. Druet et al. (1978) noted renal immunologic insufficiencies in Brown Norway rats given subcutaneous injections of mercuric chloride (100 µg/kg) for 8 to 12 weeks. 3.2.3. Chronic Toxicity 3.2.3.1. Human Chronic exposure to low levels of mercury vapor may induce immunologic glomerular disease (Goyer 1991). A number of studies have been conducted with individuals occupationally exposed to inorganic mercury compounds (mercuric oxides, mercurial chlorides, mercuric nitrate) and have been reviewed by the USAF (1990). Briefly, neuropsychological symptoms (insomnia, fatigue, headaches, etc.) and renal effects that correlated with blood mercury levels were reported for those exposed 2 years. The emotional and psychological disturbances often referred to as the "Mad Hatter Syndrome" has been attributed to inhalation of the dust or vapors of mercuric nitrate used in the making of felt hats (Clarkson 1989). Central nervous system effects including fatigue, tremors, and gingivitis have been reported for chronic exposures to mercury vapor (Goyer 1991). As exposure increases, the frequency and magnitude of muscle tremors increase and are accompanied by personality and behavioral changes (memory loss, excitability, depression, and hallucinations). Low-level chronic exposures to mercury may affect the peripheral nervous system resulting in polyneuropathies (reduced sensory and motor nerve function) and neuropsychological effects (visual alterations, sensory loss, stress) (ATSDR 1989); these effects correlate to tissue levels of 20 to 40 µg/g. Neuropsychological effects were also reported by Smith et al. (1970) for occupational exposure to mercury levels of > 0.1 mg/m3. Mercury concentrations below this value did not appear to cause observable effects. Kishi et al. (1993) reported that neurobehavioral and motor function effects persisted in ex-mercury miners more than 10 years after cessation of exposure. Several reports regarding occupational exposure of chloralkali workers to mercury vapor are available. Fawer et al. (1983) reported an increase in the frequency of intention tremors of workers exposed to mercury vapor (time-weighted-average of 0.026 mg/m3) over an average of 26 years. Piikivi and Tolonen (1989) found alterations in EEGs in workers exposed to mercury vapor for an average of 15.6 years. Piikivi and Hanninen (1989) reported adverse change in subjective measures of memory disturbance and sleep disorders in workers occupationally exposed for an average of 13.7 years. Subjectively and objectively determined alterations in autonomic function (pulse rate, blood pressure autonomic reflexes) were reported for workers exposed to mercury vapor for an average of 15.6 years. Neurobehavioral effects (motor speed, visual scanning, visuomotor coordination and concentration, visual memory, visuomotor coordination speed) were affected in individuals occupationally exposed to TWA concentrations of 0.014 mg/m3 (Ngim et al. 1992). Workers exposed to mercury vapor concentrations of 0.033 mg/m3 (range 0.005 to 0.19 mg/m3) for at least two years exhibited significantly poorer performance on neurobehavioral tests than did unexposed control subjects (Liang et al. 1993). Inhalation exposure to alkyl mercury compounds may occur during the manufacture or use of alkylmercury fungicides. The effects reported for these compounds include paresthesia of the extremities, mouth and lips, constriction of the visual field, deafness, motor incoordination and compromised reflex function. In severe cases, loss of speech and mental deterioration may occur (McComish et al. 1988). Endocrine function of the pituitary, thyroid, testes, and adrenal glands was studied in chloralkali workers exposed to mercury vapor for an average of 10 years (Bårregard et al., 1994). With the exception of inhibition of deiodination of T4 to T3, no significant effects were detected in the endocrine functions studied. Mercury vapor from dental amalgams has been identified as a major source of exposure to inorganic mercury in the general population (WHO 1991). An average mercury dose from dental amalgams has been estimated to be only 4 to 5 µg (Halbach 1995). 3.2.3.2. Animal Chronic inhalation exposure (72 to 83 weeks) of rats, rabbits, and dogs to metallic mercury vapor (0.01 mg/m3) did not produce histological evidence of renal toxicity (Ashe et al. 1953). Additional information on the chronic inhalation toxicity of inorganic mercury in animals was not available. Information regarding the toxicity of organic mercury following chronic exposure of animals was not available. |
| <Thuy_Trang_Nguyen> |
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#185
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Re: 27,2 kg thủy ngân phát tán ra môi trường, kiến nghị tẩy độc khu vực Nhà máy Rạng Đông
thằng nào đòi kỷ luật chủ tịch phường đâu, tao thấy thằng chủ tịch này nó tài hèn sức mọn, nhưng nó cũng có ích cho dân hơn cái tụi chóp bu ở trên sở đó. Má mấy thằng lol trên tp ỷ mạnh hiếp yếu, gặp tao là chủ tịch phường đó là tao nộp đơn từ chức về chạy grab cũng dc. Cho nó nhục mặt nó ra.
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| pepguard-violon |
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#186
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Re: 27,2 kg thủy ngân phát tán ra môi trường, kiến nghị tẩy độc khu vực Nhà máy Rạng Đông
Quote:
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#187
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Re: 27,2 kg thủy ngân phát tán ra môi trường, kiến nghị tẩy độc khu vực Nhà máy Rạng Đông
Hồi trước có vỡ cái nhiệt kế thôi mẹ tôi đã bắt cả nhà sang bà ở cả tuần để thủy ngân ở nhà nó bay hơi hết rồi đấy
![]() Đây 27kg
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| aVI_Faraday |
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#188
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Re: 27,2 kg thủy ngân phát tán ra môi trường, kiến nghị tẩy độc khu vực Nhà máy Rạng Đông
xàm lon, nhiệt kế thủy ngân nào?
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#189
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Re: 27,2 kg thủy ngân phát tán ra môi trường, kiến nghị tẩy độc khu vực Nhà máy Rạng Đông
Cái cặp nhiệt độ để đo nhiệt độ cơ thể ấy
![]() Cái giọt ở trong là thủy ngân đấy
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| aVI_Faraday |
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#190
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Re: 27,2 kg thủy ngân phát tán ra môi trường, kiến nghị tẩy độc khu vực Nhà máy Rạng Đông
ko phải bằng thủy ngân đâu.
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